differences between Schizophrenia and Bipolar?

Bipolar disorder is a mood disorder which effects a persons mood and is caused by chemical imbalances in the brain. In bipolar disorder a person has mood swings from depression to mania which have to last 7 days each to be diagnosed but generally last weeks or months at a time. I developed bipolar when I was 15 and diagnosed at 18, when I was depressed I would sleep constantly, cry, self harm, wouldn't leave bed for days, didn't eat, wash, dress, constantly felt suicidal, hopeless and worthless, withdrew myself from family and friends and stopped going to school. This lasted for 6 months. Then I switched into mania when I was constantly full of energy and felt like I was on top of the world I was so happy, didn't sleep for days and if I did I would sleep up to 3 hours only, went out every night abusing alcohol and drugs, got into meaningless relationship, had sex with strangers, spent every penny I had and even stole from my mum, shop lifted, racing thoughts, aggressive and irritable, psychotic when I would hallucinate and be delusional to the point I stabbed myself and almost my boyfriend, I was constantly doing one thing to the next and I nearly failed college. This lasted for 4 months. Bipolar disorder is something that is very severe and effects your everyday life. I ended up being hospitalised because I was psychotic, hallucinating and delusional. 

Schizophrenia is when a person usually experiences hallucinations, delusions and extreme paranoia which makes them lose contact with reality and not understand what is real and what is not as they believe the hallucinations and delusions are real. Other symptoms include disorganised speech, catatonia, depression, self harm, suicidal and/or homicidal thoughts, insomnia, anxiety. These symptoms have to be on going for a full 6 months before being diagnosed with schizophrenia. People who have schizophrenia usually have no idea that they are ill in any kind of way and often deny that anything is wrong with them.

My friend has schizophrenia and it started when she would say that everything tasted funny and decided she was not going ot eat anything anymore because people were trying to poison her. Then I noticed she would talk to herself in her room even though no one was there, she would sit and stare into space and randomly laugh for hours. She withdrew herself from us (her friends) and claimed we only wanted to be her friend to get inside her head and kill her and that she had new friends now. Her new friends were not real, they were voices and hallucinations in her head. She was constantly on edge and really paranoid, sometimes I would talk to her online and she would say she didn't know me but other days would say things like her friend told her I was bad and she had to stay away from me. The last time I saw her before she was hospitalised she had not slept in days and looked very tired and was very edgey and paranoid, she could not keep eye contact from fear of being killed. She had cuts from her wrist to elbow on each arm which were self inflicted and they all needed stitches. I took her home and told her parents and left. Next thing she was in her room threatening to kill herself and her parents with a knife, she was then arrested and sectioned for just under a year getting better. This all went on for 8 months before she got the help.



Bipolar - "Periods of hyperactivity followed by periods of depression." In other words, a person's mood fluctuates (changes) very quickly and without warning. In the matter of moments, the person can go from a feeling of euphoria to one of intense anger or sorrow.


Schizophrenia - A person with Schizophrenia may have one or more of the following:
Hallucinations
Hearing or seeing things that aren't there
Thinking that someone is inside their head
Claiming that someone is guiding or telling them what to do
Becoming paranoid, or believing things that aren't true or real
Difficulty speaking and organizing thoughts may result in stopping speech midsentence or putting together meaningless words, sometimes known as "word salad."


Most people consider Schizophrenia a far more severe disorder than Bipolar.

Is Nutrascience effective or not?

Nutrascience is the world’s best weight loss supplement to kick away unwanted pounds and expel perilous toxins from the body. Its natural ingredients are 100% safe in burning extra fat & calorie. So don’t be confused just try it….

Yup,you can say it's very effective and totally risk free product for slim.And you know i got slim body through NutraScince so you should go with this..so now decision on your hand what do you want???


The ingredients in the patch are absorbed by the body, which then reaps the full benefits for increased metabolism, feeling fuller for longer and all of this leads to losing weight.


Yes Nutrascience very effective product. Nutrascience god product for weight loss.


Yes, its really effective.. because i use it and satisfy with it... that's all.

Smells Like Parkinson’s Disease

Parkinson’s disease has always been primarily seen as a movement disorder resulting in symptoms of shaking, tremors, rigidity, and trouble walking. Interestingly, however, at least 90% of patients with Parkinson’s experience either loss or decreases in the sense of smell. Studies have shown that problems with olfaction actually generally precede the onset of other motor symptoms. Most people are not personally aware of changes in their olfactory acuity, but the increasing range and prevalence of smell tests offer a quick, easy, cheap, and non-invasive diagnostic test, as well as a measure of disease progression. In addition, the shift of focus for researchers from Parkinson’s as a motor disorder to a more global neurodegenerative disorder allows consideration of new paradigms about the causes and disease progression.
The cellular basis of olfactory dysfunction in Parkinson’s remains an enigma. Post-mortem studies have confirmed shrinkage of the olfactory bulb, but this fails to shed light onto the root causes as it only demonstrates the end effect. Experimental models of Parkinson’s have demonstrated various results such as protein aggregation in the olfactory bulb, changes in levels of neurotransmitters, microglial activation, and loss of cells in the olfactory bulb. However, as all of these effects are inter-related, none of these clarify the actual initial cause of damage.
Many of the hypotheses as to why olfactory dysfunction occurs and precedes other symptoms remain grounded in the long-held paradigm of Parkinson’s as a motor disease caused by the loss of dopaminergic neurons in the substantia nigra. For example, in some experimental models an increase in dopamine was found in the olfactory bulb. The researchers suggested this occurs as a compensatory mechanism in response to the loss of dopamine in the substantia nigra. As it has also been shown that sense of smell is particularly vulnerable to changes in dopamine, excess dopamine in the olfactory bulb would, thus, lead to olfactory dysfunction.
However, a number of other theories have been proposed suggesting that perhaps the olfactory bulb is the first brain structure to exhibit signs of damage because Parkinson’s could be caused by respiratory viruses or inhaled toxins that enter the brain through the nose. The cause of Parkinson’s has not been conclusively determined. And while a genetic component has been uncovered, the causes are clearly much more complex with various environmental factors involved. A number of studies have been performed demonstrating Parkinson’s-like symptoms following exposure to viruses, heavy metals, and pesticides in experimental models. Epidemiological studies have also linked pesticides exposure to an increased risk of Parkinson’s. It is certainly an interesting hypothesis that inhaled toxins could cross the blood brain barrier, and that the damage in Parkinson’s could begin first in the olfactory bulb and then spread from there to the substantia nigra. In addition, as the olfactory bulb is heavily involved in adult neurogenesis, any damage to this structure could severely limit the brain’s ability to repair itself by replenishing damaged neurons with new ones. Perhaps, then, Parkinson’s disease does not depend on a single source of damage, but rather multiple insults occurring. For example, genetically induced damage to the dopaminergic neurons in the substantia nigra combined with inhaled toxins damaging the olfactory bulb could, together, cause Parkinson’s, while one or the other would be insufficient.